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Also, when con-sidering the clinical relevance of Pin1 deregulation it should be kept in mind thatPin1 may also collaborate with mechanisms of tumor suppression as for examplep53- or p73- induced apoptosis [ 67 , 73] as well as degradation of CMYC [ 74 ] andCCNE [ 75]. Almost all patients developedantibodies to galsulfase [ 36]

Almost all patients developedantibodies to galsulfase [ 36]. One of these cells that werementioned by the authors of the histologic study of RCC DC phenotypes was the presence ofnumerous CD11c+ macrophages (Middel 2010)

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